Venue Templates
Format Cell Press–style Summary, Highlights, and eTOC blurbs when drafting or polishing manuscripts with an agent.
Overview
Venue-templates is an agent skill for the Build phase that supplies Cell Press Summary, Highlights, and eTOC blurb examples and formatting patterns for scientific manuscripts.
Install
npx skills add https://github.com/k-dense-ai/scientific-agent-skills --skill venue-templatesWhat is this skill?
- Complete Cell Press Summary example capped at 150 words with senescence/FOXO4 narrative structure
- Highlights bullets with ≤85 characters per line and consistent scientific tone
- eTOC blurb patterns aligned with Cell Press venue expectations
- Copy-paste-ready blocks for agent-assisted manuscript polish before journal submission
- Summary max 150 words in the documented example
- Highlights bullets ≤85 characters each
Adoption & trust: 536 installs on skills.sh; 27.6k GitHub stars; 3/3 security scanners passed (skills.sh audits).
What problem does it solve?
You have draft results but no venue-correct Summary, Highlights, or eTOC text that meets Cell Press length and style rules.
Who is it for?
Researchers or agent builders preparing Cell Press–family submissions who want consistent abstract and highlight formatting from SKILL.md examples.
Skip if: Builders who need generic APA/IEEE templates, non–Cell Press venues, or automated peer review—not venue-specific prose shells.
When should I use this skill?
Drafting or revising Cell Press Summary, Highlights, or eTOC text for a scientific manuscript.
What do I get? / Deliverables
After the skill runs, you get copy-ready Summary, Highlights, and eTOC blocks sized for Cell Press submission workflows.
- Cell Press–style Summary paragraph
- Highlights bullet list
- eTOC blurb copy
Recommended Skills
Journey fit
Manuscript packaging (abstract, highlights, TOC blurb) is documentation work done while building and finalizing a research artifact for submission. The skill is canonical shelf in docs because it outputs structured prose templates, not lab analysis or distribution campaigns.
How it compares
Use for publisher-specific prose templates instead of ad-hoc chat paraphrasing of journal guidelines.
Common Questions / FAQ
Who is venue-templates for?
Solo builders and small teams using scientific agent skills who submit to Cell Press venues and need standardized Summary, Highlights, and eTOC blurbs.
When should I use venue-templates?
Use during Build/docs when finalizing a paper package, or before launch-style distribution to editors, whenever Cell Press element limits apply.
Is venue-templates safe to install?
It is documentation-only with no shell or network requirements; review the Security Audits panel on this Prism page before installing from any source.
SKILL.md
READMESKILL.md - Venue Templates
# Cell Press Summary, Highlights, and eTOC Examples Examples of Cell Press-specific elements including Summary (abstract), Highlights, and eTOC blurb. --- ## Complete Example 1: Senescence and Aging ### Summary (150 words max) ``` Cellular senescence is a stress response that prevents damaged cell proliferation but can drive tissue dysfunction through the senescence- associated secretory phenotype (SASP). How senescent cells resist apoptosis despite expressing pro-apoptotic p53 has remained unclear. Here, we identify FOXO4 as a pivotal mediator of senescent cell viability. FOXO4 is highly expressed in senescent cells and directly interacts with p53, retaining it in the nucleus and preventing p53-mediated apoptosis. A cell-permeable peptide that disrupts FOXO4-p53 interaction selectively induces p53 nuclear exclusion and apoptosis in senescent cells without affecting proliferating cells. In vivo, this FOXO4 peptide neutralizes doxorubicin-induced senescent cells and restores fitness, fur density, and renal function in naturally aged mice. These findings establish FOXO4-mediated p53 sequestration as a senescence-specific survival pathway and demonstrate the therapeutic potential of targeted senescent cell elimination. ``` ### Highlights (≤85 characters each) ``` • FOXO4 is selectively upregulated in senescent cells and binds p53 • FOXO4-p53 interaction retains p53 in the nucleus, preventing apoptosis • A FOXO4-targeting peptide induces apoptosis specifically in senescent cells • FOXO4 peptide treatment restores fitness and organ function in aged mice ``` ### eTOC Blurb (30-50 words) ``` Baar et al. identify FOXO4 as a critical mediator of senescent cell survival through p53 sequestration. A peptide disrupting FOXO4-p53 interaction selectively eliminates senescent cells and restores tissue function in aged mice, establishing proof-of-concept for targeted senolytic therapy. ``` ### In Brief (1 sentence) ``` A FOXO4-targeting peptide selectively eliminates senescent cells by releasing p53, restoring tissue function in aged mice. ``` --- ## Complete Example 2: Genome Organization ### Summary (150 words max) ``` The three-dimensional organization of chromosomes within the nucleus influences gene expression, DNA replication, and genome stability. Phase separation has emerged as a potential mechanism for organizing nuclear contents, but whether condensates can shape chromosome structure in vivo remains unknown. Here, we show that the transcriptional coactivator BRD4 forms liquid-like condensates at super-enhancers that organize associated chromatin into hub structures. Optogenetic induction of BRD4 condensates is sufficient to remodel chromosome topology and activate transcription within minutes. Conversely, disruption of BRD4 condensates with the small molecule JQ1 dissolves chromatin hubs and rapidly silences super-enhancer-controlled genes. Single-molecule tracking reveals that condensate formation increases the local concentration of transcription machinery 100-fold, explaining the transcriptional potency of super-enhancers. These results establish phase separation as a mechanism for chromatin organization and transcriptional control with implications for understanding and targeting oncogenic super-enhancers. ``` ### Highlights ``` • BRD4 forms liquid condensates at super-enhancers in living cells • BRD4 condensates organize chromatin into transcriptionally active hubs • Optogenetic condensate induction rapidly remodels chromatin topology • Condensates concentrate transcription machinery 100-fold locally ``` ### eTOC Blurb ``` Sabari et al. demonstrate that BRD4 forms phase-separated condensates at super-enhancers that organize chromatin into hub structures and concentrate transcription machinery. Optogenetic manipulation reveals that condensate formation directly drives chromatin remodeling and transcriptional activation. ``` --- ## Complete Example 3: Metabolism and